By C.M. Poser

ISBN-10: 1850709467

ISBN-13: 9781850709466

This can be a thoroughly new, completely up to date colour atlas-textbook of a number of sclerosis (MS). the 1st of its sort, it bargains unique worth for its illustrations and instructive textual content at the very most modern diagnostic approaches for MS. The e-book opens with introductory and history info after which covers the pathogenesis of MS concerning alteration of the blood-brain barrier. The part on prognosis covers IgG and oligoclonal bands within the cerebrospinal fluid; evoked power reviews, together with visible, brainstem auditory, and somatosensory; MRI (magnetic resonance imaging), together with MRI differential analysis concerning ADEM, MDEM, Lyme disorder, and HTLV-I spastic paraparesis; and puppy (positron emission tomography) and SPECT (single photon emission tomography). Concluding sections conceal the gross, microscopic, and electronmicroscopic pathology of MS and the visible method in MS, regarding optic and retrobulbar neuritis; fundoscopy, and visible fields. comprises bibliographic re ferences and index.

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Extra resources for An Atlas of Multiple Sclerosis

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Brain Dev 1996;18:412–4 Poser C, Roman G, Vernant J-C. Multiple sclerosis or HTLV-I myelitis? Neurology 1990;40:1020–2 Woyciechowska J, Dambrozia J, Chu A, et al. Correlation of oligoclonal IgG bands and viral antibodies in twins with multiple sclerosis. , eds. Virology and Immunology in Multiple Sclerosis. Berlin: Springer-Verlag, 1987:45–9 Pozzilli C, Bernardi S, Mansi U, et al. Quantitative assessment of blood–brain barrier permeability in multiple sclerosis using 68-Ga-EDTA and positron emission tomography.

The MRIs (right), obtained approximately 2 months later, after recovery, show few areas of increased signal intensity (AISI). With the possible exception of the enhanced area at the superior angle of the left ventricle, none of the areas of BBB alteration seen on CT are evident on MRI. Their disappearance so soon after clinical recovery suggests that the contrast enhancement represents edema rather than demyelination, as remyelination does not occur within such a short period of time. , 1987; reproduced with the permission of the American Society of Radiology ©1998 CRC Press LLC Figure 22 The disease process in multiple sclerosis can be compared with volcanic island chains such as the Hawaiian Islands, where only the tips of the volcanic structures protrude above sea level while unseen activity continues below Figure 23 Unstained gross coronal section of brain.

La perméabilité des vaisseaux cérébraux. IV. Démyélinisation expérimentale provoquée par des substances agissant sur la barrière hématoencéphalique. Acta Neurol Belg 1966;66:247– 62 Haegert D, Swift F, Benedikz J. Evidence for a complex role of HLA class II genotypes in susceptibility to multiple sclerosis in Iceland. Neurology 196;46:1107–11 Jahnke U, Fischer E, Alvord E. Sequence homology between certain viral proteins and proteins related to encephalomyelitis and neuritis. Science 1985;229:282–4 Jansen H, Willemsen A, Sinnige L, et al.

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An Atlas of Multiple Sclerosis by C.M. Poser

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